Distinct molecular etiologies of male and female hepatocellular carcinoma

Keyphrases

• Hepatocellular Carcinoma 100%
• Molecular Etiology 100%
• Sex-specific 60%
• Tumor 50%
• Expression Quantitative Trait Loci (eQTL) 30%
• Sex Differences 30%
• Signaling Pathway 20%
• Gene Expression 20%
• Differential Effects 20%
• Dysregulation 20%
• Cancer Mortality 20%
• Differential Expression Analysis 20%
• QTL Analysis 20%
• Pathway Enrichment 20%
• In Cancer 10%
• Clinically Significant 10%
• Etiology 10%
• Male Bias 10%
• Gender Bias 10%
• Gender Analysis 10%
• Germline Variants 10%
• Tumor Subtype 10%
• Regulatory Change 10%
• Response to Treatment 10%
• Cancer Causes 10%
• Poor Response 10%
• Apoptosis 10%
• Biological Mechanisms 10%
• Genetic Effects 10%
• Epigenetic Regulation 10%
• Relevant Genes 10%
• Phosphoinositide 3-kinase (PI3K) 10%
• Transcription Regulation 10%
• Sex-stratified 10%
• Cancer Occurrence 10%
• AKT1 10%
• Rap1 10%
• Tumor Gene Expression 10%
• Egfr 10%
• DAP12 10%
• PPAR Signaling Pathway 10%
• Fgfr 10%
• IL-2 Signaling 10%

Biochemistry, Genetics and Molecular Biology

• Gene Expression 100%
• Expression Quantitative Trait Loci 100%
• Sex Difference 100%
• Cell Cycle 33%
• Germ Cell 33%
• Germline 33%
• Transcription 33%
• Expression Analysis 33%
• Epidermal Growth Factor Receptor 33%
• Genetic Determinism 33%
• Cancer Mortality 33%
• Genetic Regulation 33%
• Peroxisome Proliferator-Activated Receptor 33%
• Tumor Gene 33%
• Protein Kinase B 33%
• Interleukin 2 33%
• Rap1 33%
• Programmed Cell Death 33%