Abstract
We consider four mechanisms by which apparent discontinuities in the distribution of antisociality could arise: (1) executive genes or hormonal systems, (2) multiplicative interactions of predisposing factors, (3) environmental tracking into a limited number of social roles, and (4) cross-generational gene-environment interactions. A more explicit consideration of complex self-organizing dynamic systems may help us understand the maintenance of antisocial subpopulations.
Original language | English (US) |
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Pages (from-to) | 560-561 |
Number of pages | 2 |
Journal | Behavioral and Brain Sciences |
Volume | 18 |
Issue number | 3 |
DOIs | |
State | Published - 1995 |
ASJC Scopus subject areas
- Neuropsychology and Physiological Psychology
- Physiology
- Behavioral Neuroscience